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Abstract
To elucidate the correlation between CKLF-like marvel transmembrane domain containing member (CMTM5) gene and the risk of in-stent restenosis (ISR) with coronary artery disease (CAD) patients and to detect the effects and mechanisms of CMTM5-stimulated genes on human vascular endothelial cells (ECs) proliferation and migration.
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MeSH headings
Chemokines, Coronary Artery Disease, Coronary Restenosis, Drug-Eluting Stents, Endothelial Cells, Humans, MARVEL Domain-Containing Proteins, Phosphatidylinositol 3-Kinases, Tumor Suppressor Proteins
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