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MS Elias, HA Long, CF Newman, PA Wilson, A West, PJ McGill, KC Wu, MJ Donaldson and NJ Reynolds
Abstract
Atopic eczema (AE) is characterized by skin barrier and immune dysfunction. Null mutations in filaggrin (FLG), a key epidermal barrier protein, strongly predispose to AE; however, the precise role of FLG deficiency in AE pathogenesis remains incompletely understood.
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Concepts
Proteins, DNA, Autoimmune diseases, Immune system, Proteomics, Psoriasis, Eczema
MeSH headings
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