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A Kumar, BA Stoica, DJ Loane, M Yang, G Abulwerdi, N Khan, A Kumar, SR Thom and AI Faden
Abstract
Local and systemic inflammatory responses are initiated early after traumatic brain injury (TBI), and may play a key role in the secondary injury processes resulting in neuronal loss and neurological deficits. However, the mechanisms responsible for the rapid expansion of neuroinflammation and its long-term progression have yet to be elucidated. Here, we investigate the role of microparticles (MP), a member of the extracellular vesicle family, in the exchange of pro-inflammatory molecules between brain immune cells, as well as their transfer to the systemic circulation, as key pathways of inflammation propagation following brain trauma.
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Concepts
Cerebellum, Systemic inflammatory response syndrome, Neuron, Member of Parliament, Inflammation, Immune system, Brain, Traumatic brain injury
MeSH headings
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